Thyroid paradox

ricwally

There are two distinct means with which a cell can be reproduced. The first method is by way of
the cell replicating itself as outlined within that cell’s DNA. The second method is the slightly
altered and inferior procedure of the body’s own immune system rapidly reproducing the
surrounding tissues in an attempt to heal over an area by way of ‘scar tissue’.
The immune system actually has three distinct components;
i) to ‘identify’ foreign antigens that are deemed to be ‘enemies of the body’
ii) to ‘destroy’ these enemies of the body; and
iii) to ‘repair’ any damage that may have occurred during this onslaught.
This article will be focusing on this ‘repair’ aspect of the immune system, which expressed
simply, is the bodies ability to promote rapid cell division (the formation of scar tissue) to
quickly heal over breaks, wounds or openings in the skin. The mechanism that starts this process
is triggered when the body experiences some form of trauma. But once this process is started,
there also needs to be some mechanism in place to inform the body of when the healing process
has been completed. That is to say, the body must be made to know when the rapid formation of
scar tissue is no longer required, so that the immune system can cease this elevated activity, and
restore itself to the level of activity that existed prior to the trauma. It does not require too much
imagination to realize that the inability to shut off this ‘repair process’, would result in a situation comparable to that which we presently attribute to cancer. For example, a trauma to the breast would trigger the immune response of repairing any tissues that may have been damaged.
If the immune system lacked the ability to know when this process was completed, it would go
on to repair the tissues in the breast, and a tumor resembling the scar tissue process(firmer
density, different collagen alignment, different pigment, etc.) would be the result. Similarly, if a faulty immune system were to commence this healing process without there first being a
requirement for it, then this too would result in an activity that could fit the description of cancer.
Since there are two distinct ways in which a cell can be reproduced, we should be considering
both of these scenarios as possible explanations that might be the cause when something goes
wrong. Thus far, only the DNA model has been investigated as being the cause of this affliction.
This article will now examine scar tissue as a possible cause of this non-requested cell
replacement that we commonly refer to as ‘cancer’.
The immune system has in its arsenal, the ability to inflame an area with increased blood flow,
and stimulate the neighboring cells into rapidly reproducing themselves, in order to quickly seal
over an opening in the skin, which stops blood loss and prevent foreign antigens from entering
the body by way of this new opening. It is known that this process is set in motion when the body
experiences some form of trauma. When we examine this activity more closely, it can be
observed that there are similarities between cancerous activity; and the inflammation and
formation of scare tissue. When we can readily observe scar tissue, as in the case of skin surface
scars, we can readily detect that this is an altered form from that of the surrounding tissue.
Because it was manufactured rapidly, and by a different process than that of normal tissue
replacement (normal cell division, as outlined in that cell’s DNA), it has different
characteristics. For example, scar tissue made from skin cells has a distinct appearance with a
smoother surface, firmer density, (described as a waxy appearance) and a different pigment from
that of the surrounding tissue. A clinical definition is as follows:

Scar tissue formation is a ubiquitous feature of adult wound healing, with the resulting
repair both functionally and cosmetically inferior to normal skin. At microscopic level,
the main difference between scar and normal tissue is in the alignment pattern of the
collagen fibers of which they are composed.
www.google.com final report on Grant GR/K71394
Mathematical Model of Scar Tissue
This excerpt acknowledges that there are indeed two distinct ways that a cell can be reproduced.
Firstly, by the well understood way of the cell’s natural means of replicating itself as outlined in
the cell’s DNA code, which is referred to above as ‘normal’ cell replacement, and secondly, by a
less obvious, and less understood process whereas the bodies immune system triggers the cells
into this slightly altered scar tissue. Note that this second means of cell replacement (scar tissue)
is described as “functionally and cosmetically inferior”. The rapid growth, and the inferior
quality of tissues are two attributes shared by both the tissues manufactured by the immune
system, and the tissues manufactured by cancer cells. The primary means of cell replacement
does not have attributed to it, these inferior qualities that the immune system replacement method
has. In fact, the purpose of a Burn Unit is to hinder the bodies tendency to rapidly heal over the
burned area with scar tissue, when the trauma of a burn has set off this immune response, and
allow the slower process (but cosmetically superior) of natural cell replacement to have enough
time to heal the area.
The easiest cancers to observe are the surface cancers. Notice that Basil Cell Carcinoma has all
of the characteristics of scar tissue (smother, denser, waxy.). This common skin cancer could
conversely be described as a slow formation of scar tissue that is both unnecessary, and
unyielding. This cancer is not considered to be a dangerous cancer because it is slow growing and
easily removed surgically. With this new model, we could regard this cancer to be different in
that; although it has the cell division element,( cells being divided by either faulty DNA, or a
faulty immune system) it does not have the accompanying blood supply (inflammation) which is
necessary to support the existence of these newly formed cells. Note that the shape of the basil
cell carcinoma would indicate that it can only grow to a size that can be supported by the existing
blood supply, and as it grows, the center cells cannot receive oxygen or nutrients, and as a result,
these center cells die off, leaving a hollow in the middle. If this tumor were to have its own blood
supply, it would become considerably more dangerous.
Both the ‘DNA theory’, and this ‘Scar Tissue theory’ are able to adequately account for the
cancer cells having shared characteristics from the ‘host’ cells, however the latter theory becomes
much more complex by virtue of the fact that it must also account for the modification of the
existing blood supply. The ‘Scare Tissue theory’, is not required to account for the
accompanying increased blood supply, because the same elements that brought about the
reproduction of the cells, also caused the accompanying blood supply (inflammation). Both of
these events are normal functions of the immune system responding to a trauma. The DNA
theory, must further account for the presence of the accompanying blood flow to support the life
of these newly generated cells. It is comprehensible how the DNA of an individual cell might go
astray, and start to reproduce itself repeatedly, but this event would be limited to grow only to the
size that could be supported by the existing blood supply. It should yield a ‘pea’ sized growth. If
this chain of events were to occur, the first step would be the cell replicating itself. It is
reasonable to expect that there would be a number of occurrences in which this chain of events
did not complete itself. That is to say, there should be occurrences in which the cell did
reproduce itself, but the accompanying blood supply did not happen. The scientific community
acknowledges the need to address the blood supply issue, and with great difficulty they have
postulated a complex chain of events that is both mathematically and logically absurd. We are
told that these cancer cells take on an ‘immortal’ status, and acquire the ability to ‘disguise’
themselves, and ‘recruit allies’ in there defense, and a multitude of other special powers that are
attributed only to cancer cells. When you examine this supernatural chain of events, and the
obstacles that the cancer must overcome, and the safeguards that are in place to prevent these
occurrences from happening the way they are described, you would wonder about the
mathematical likelihood of this occurring even once. It requires much less credence to simply
hold that the immune system is causing the lawless proliferation of growth, (since it is its job to
do so,) and the immune system is also supplying the essential blood supply to support this new
growth, by way of ‘inflammation’. If we make this simple adjustment in our model for
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