Feb 19, 2013 - 4:15 pm
So I am a recent diagnosis of Rectal Cancer. I did 6 weeks of chemo/radiation. The radiation was really horrible as some of you probably know, unfortanately.
I am currently feeling the best I've felt since I was dianosed though. I had a tumor blocking almost 60% of my colon at the time I went to the emergency room. The reason I got so bad was because of the military failing to diagnose me properly. That's a another forum topic for later though.
I was really interested in knowing if anyone has experimented with any other treatments? The only option the mainstream cancer industry is giving me, rectum removal and colostomy bad for life. Don't plan on doing that. I have done lots of research and have been experimenting with everyting I feel would be beneficial. Stuff like Alluminum free sodium bicarbonate, turmeric, ginger, lemon, teas, vegan diet, juicing veggies, wheat grass shots, just to name a few. Now I feel really good but the doctors say that is temporary, That the tumor will not only grow back, but grow back faster because of the chemo/radiation. I find that to be very rediculous. How does the official billion billion dollar cancer industry treatment for cancer, cause it to grow back faster. Something suspect about that. Not to mention you're consider a survior in their eyes if you live 5 years after diagnosis. So even if you die at 5 years and 3 months after diagnosis, you're still a "cancer survior".
My next alternative treatment is going to be moving to OR and getting my medical cannabis card. Then I plan on getting high concentrated cannabis oil suppositories, ingesting the cannabis oil orally, and smoking/vaporizing it. Everything that I've read shows this helps not only with the pain but healing your body as well. I'm no doctor and have only read about testominials and what not but if you go to http://www.cancer.gov/cancertopics/pdq/cam/cannabis/healthprofessional/page4 this shows very promising studies, research, experiments in the medical cannabis field. If you read over all the tests they do in mice and rats, it basically shows results like shrinking tumors, having cancer cells self destruct while keeping normals cells healthy, and much more. Just have to read through it all.
So if anyone has any experiences with any alternative treatments, please share them.
For all the lazy people that don't like to copy and paste links, here is some info from cancer.gov in regards to cannabis oil and anti-tumor effects.
Hope this helps.
Please share any info you have.
One study in mice and rats suggested that cannabinoids may have a protective effect against the development of certain types of tumors. During this 2-year study, groups of mice and rats were given various doses of THC by gavage. A dose-related decrease in the incidence of hepatic adenoma tumors and hepatocellular carcinoma was observed in the mice. Decreased incidences of benign tumors(polyps and adenomas) in other organs (mammary gland, uterus, pituitary, testis, and pancreas) were also noted in the rats. In another study, delta-9-THC, delta-8-THC, and cannabinol were found to inhibit the growth of Lewis lung adenocarcinoma cells in vitro and in vivo . In addition, other tumors have been shown to be sensitive to cannabinoid-induced growth inhibition.[5-8]
Cannabinoids may cause antitumor effects by various mechanisms, including induction of cell death, inhibition of cell growth, and inhibition of tumor angiogenesis invasion and metastasis.[9-12] One review summarizes the molecular mechanisms of action of cannabinoids as antitumor agents. Cannabinoids appear to kill tumor cells but do not affect their nontransformed counterparts and may even protect them from cell death. These compounds have been shown to induce apoptosis in gliomacells in culture and induce regression of glioma tumors in mice and rats. Cannabinoids protect normal glial cells of astroglial and oligodendroglial lineages from apoptosis mediated by the CB1 receptor.
The effects of delta-9-THC and a synthetic agonist of the CB2 receptor were investigated in hepatocellular carcinoma (HCC). Both agents reduced the viability of hepatocellular carcinoma cellsin vitro and demonstrated antitumor effects in hepatocellular carcinoma subcutaneous xenografts in nude mice. The investigations documented that the anti-HCC effects are mediated by way of the CB2 receptor. Similar to findings in glioma cells, the cannabinoids were shown to trigger cell death through stimulation of an endoplasmic reticulum stress pathway that activates autophagy and promotes apoptosis. Other investigations have confirmed that CB1 and CB2 receptors may be potential targets innon-small cell lung carcinoma  and breast cancer.
An in vitro study of the effect of CBD on programmed cell death in breast cancer cell lines found that CBD induced programmed cell death, independent of the CB1, CB2, or vanilloid receptors. CBD inhibited the survival of both estrogen receptor–positive and estrogen receptor–negative breast cancercell lines, inducing apoptosis in a concentration-dependent manner while having little effect on nontumorigenic, mammary cells.
CBD has also been demonstrated to exert a chemopreventive effect in a mouse model of colon cancer. In the experimental system, azoxymethane increased premalignant and malignant lesions in the mouse colon. Animals treated with azoxymethane and CBD concurrently were protected from developing premalignant and malignant lesions. In in vitro experiments involving colorectal cancer cell lines, the investigators found that CBD protected DNA from oxidative damage, increased endocannabinoid levels, and reduced cell proliferation.
Another investigation into the antitumor effects of CBD examined the role of intercellular adhesion molecule-1 (ICAM-1). ICAM-1 expression has been reported to be negatively correlated with cancermetastasis. In lung cancer cell lines, CBD upregulated ICAM-1, leading to decreased cancer cell invasiveness.
In an in vivo model using severe combined immunodeficient mice, subcutaneous tumors were generated by inoculating the animals with cells from human non-small cell lung carcinoma cell lines. Tumor growth was inhibited by 60% in THC-treated mice compared with vehicle-treated control mice. Tumor specimens revealed that THC had antiangiogenic and antiproliferative effects. However, research with immunocompetent murine tumor models has demonstrated immunosuppression and enhanced tumor growth in mice treated with THC.[21,22]
In addition, both plant-derived and endogenous cannabinoids have been studied for anti-inflammatoryeffects. A mouse study demonstrated that endogenous cannabinoid system signaling is likely to provide intrinsic protection against colonic inflammation. As a result, a hypothesis that phytocannabinoids and endocannabinoids may be useful in the risk reduction and treatment of colorectal cancer has been developed.[24-27]